HD & CsV

[elf]

Extracts from a French PhD published in 2005 (http://revmedvet.com/2005/RVM156_138_147.pdf), sorry if the translation is not accurate I used a translator:

“E - The food is not however the only factor in
cause. The genetics remains in spite of very dominating.

It is noted that the frequency and the gravity of the DCF are
all the more important as the parents are themselves
more reached. The crossing of unscathed parents of dysplasy
give subjects among which percentage of dysplasic
is weak. This percentage increases when them
parents are reached of light dysplasy; it is even more
raised if the parents present a severe dysplasy [23, 34].
The conditions of breeding, in particular the food, do not have
that an incomplete action and cannot thwart completely
effects of the genetics. Thus, LUST [22] showed
that if the control of the food could prevent the development
DCF in dogs resulting parents phénotypiquement
unscathed or affected of light dysplasy,
a food restriction cannot prevent the dysplasy
at products whose parents have a moderate dysplasy
with severe. “


“Control of the composition of food

After the introduced quantity, the second factor on which
an intervention is possible is the composition of food
proposed. This control relates in particular to greases which
mainly determine the energy density of food,
calcium and proteins, although an excess of these
last in the dog, unlike the other species,
do not involve effects prejudicial on the metabolism of
calcium or the development of the skeleton [31]. “


“If the food is suitable, a calcic complement
is not only useless but also prejudicial. One should not
not to transpose to the food of the dog this saying of
popular wisdom according to which “abundance of good cannot
not to harm”, considering that the calcium excess will be eliminated.
However, it was shown, in various species of which the dog,
that at the very young subjects, the absorption of calcium is
directly connected to the quantity present in the food
[13].

In short, for the dogs at the risk, it is recommended of
to choose a food whose characteristics are the following ones
[31]:
- Energy: 3,2 to 3,8 kcal/g (13,4 to 15,9 kj/g)
- Grease: not more than 12% ms
- Calcium: from 0,7 to 1,2% ms
- Proteins: from 22 to 32% ms "


“Zootechnical

If the food is an important risk factor, it
is that for the genotypes dysplasic and not for
others [5]. By the search for a slow growth rate or
at least adapted better to the race, the food is likely
to mask laxity, dysplasic expression of the genotype
: the animal will become a negative forgery [6, 16]. Admittedly one
will not mask all the dysplasies, but it will result from it
larger errors on the evaluation of the genetic statute
reproducers. “


“That brings moreover to raise another question: with
which period of the growth overfeeding is
more prejudicial as for the appearance of the DCF? All
period of growth, of the birth in 12 months is in fact
sensitive, but two periods appear particularly
important:
- the first 60 days according to HAZEWINKEL [13] but
also LUST [21] which highlighted it on pups born
by Caesarean.
- the period from 3 to 8 months as the many ones attest it
authors and in particular between 2nd and the 4th month,
when the process of ossification is with its maximum. “


“It is mainly the excess of energy contribution which is
prejudicial, especially if it is combined with excesses of contribution
calcic or with ionic imbalances of the ration. “